While we are familiar with the Propionate ester the remaining three esters that create Sustanon-250 are almost always found as part of a mixture or compounded anabolic androgenic steroid .
Developed by Organon, the original idea behind Sustanon-250 was to provide a testosterone form well-suited for hormone replacement therapy that would only needed to be administered once every few weeks and for all intense purposes the idea was a success. For the performance enhancing athlete Sustanon-250 can be a fine choice but the idea of injecting only once or twice a month is not applicable here. As a performance enhancer this testosterone like all forms will need to be administered on a more frequent basis. This mixture carries with it two fast, short esters, Propionate and Pheylpropionate, a longer more moderate ester Isocaproate and the very slow and long Decanoate ester. In order to keep testosterone levels stable and at their peak most athletes will inject Sustanon-250 at a minimum of every 3 days and more commonly every other day for optimal results.
For more info see: Sustanon-250
Good question – I think I’m taking 150 every 3 weeks. I will def ask about injecting myself – just concerned about the cost. I initially started becuz of sexual issues…lower sex drive. At one point I was going every 2 weeks and wanted to hump everything in site, and as a married man that wasn’t good, so I purposely scaled back to every 3 weeks. That said, my overall concern is how long is it safe to say on this stuff. I guess its safe until it’s not, but that’s just scary. I’ve gone to 3 different urologists and they never have a straight answer. I’ve now been off my shots for about 2 months….doing a little test drive to see how much of a difference it really is making…and I’m just now starting to feel a loss in strength and endurance in my workouts, BUT overall not too much else. Anyway, that’s what prompted to me do a search for “possible” alternatives.
thanks for your advice – appreciate it.
5α-Reductase inhibitors like finasteride and dutasteride can slightly increase circulating levels of testosterone by inhibiting its metabolism .  However, these drugs do this via prevention of the conversion of testosterone into its more potent metabolite dihydrotestosterone (DHT), and this results in dramatically reduced circulating levels of DHT (which circulates at much lower relative concentrations).   In addition, local levels of DHT in so-called androgenic (5α-reductase-expressing) tissues are also markedly reduced,   and this can have a strong impact on certain effects of testosterone.   For instance, growth of body and facial hair and penile growth induced by testosterone may be inhibited by 5α-reductase inhibitors, and this could be considered undesirable in the context of, for instance, puberty induction .   On the other hand, 5α-reductase inhibitors may prevent or reduce adverse androgenic side effects of testosterone like scalp hair loss , oily skin , acne , and seborrhea .  In addition to the prevention of testosterone conversion into DHT, 5α-reductase inhibitors also prevent the formation of neurosteroids like 3α-androstanediol from testosterone, and this may have neuropsychiatric consequences in some men.